Epilepsi(frånlatinepilepsia, frångrekiskaἐπιληψία, frånepilambanein, att anfalla, gripa tag, frånlambanein, att ta.
FALLANDE-SOT.(förr ofta ss. skilda ord: (den)fallande sot(en), t. ex.b. OLAVI14 a(1578), N. RoSÉN V. ROSENSTEIN Alm.(Gbg) 1760, s. 28. fallande- VarR 15(1528) osv.; fallan- ALMQVISTAmor.165(1822, 1839),DENS.Mål.101(1840); jfröstergren(1920))[fsv.falandissot, the fallande sot; jfr för övr. under -SJUKA] KRAMPA,sbst.¹,r. l. f-;best.-an: pl.-orl.-er(jfr ovan).(kramp1621 osv.krampac.,,,1650,,,,1779,,.,krampe1538-1902)[fsv.kramp, krampa, krampe, sv. dial.krampe, krappe, motsv. d.krampe; av mnt.krampe(fsax.kramp(o)); jfr holl.kramp, t.krampf(fht.krampf(o)), ävensom eng.crampo. fr.crampe(båda lånade från kontinentalgerm. spr.); substantiveringavkramp, adj., eg. verbaladj. till KRYMPA] [KRAMP.sbst1 0] sjukligt tillstånd bestående i av vilranoberoende, häftiga muskelsammandragningar; förr äv. i sg. best. ss. sjukdomsnamn; äv. oeg., i fråga om krampaktigt ihållande grepp l. rörelser.fåkramp, förr äv.krampen. kramp i benet, ifing-rarna. Klonisk, tonisk kramp (med.).varrerv16(1588).på sängen iaghligger, som en steen, / Krampen draarmighall tijdha.forsiusspec.a8a (1620).SalvieWijneller öl.. förtager Krampe.IERICIColerus1:282 (c.(1645).den goda gossen gick bara ut att bada i Hellesponten, fick krampen och drunknade.
2.1 Übersicht 2.1.1 Fokale Anfälle 2.1.2 Generalisierte Anfälle 2.1.3 Nicht klassifizierbare epileptische Anfälle 2.2 Allgemeines zum Anfallsverlauf 2.2.1Aura 2.2.2 Status epilepticus 2.2.3 Terminalschlaf 2.2.4 Multiple Anfallsformen 3 Ursachen von Epilepsie 3.1 Pathophysiologie 3.2 Genetische Befunde bei Epilepsien
Generalized seizures Tonic clonic(in any combination) Absence Typical Atypical Absence with special features Myoclonic absence Eyelid myoclonia Myoclonic Myoclonic Myoclonic atonic Myoclonic tonic Clonic Tonic Atonic Focal seizures Unknown Epileptic spasms
Generalized seizures Tonic clonic(in any combination) Absence Typical Atypical Absence with special features Myoclonic absence Eyelid myoclonia Myoclonic Myoclonic Myoclonic atonic Myoclonic tonic Clonic Tonic Atonic Focal seizures Unknown Epileptic spasms Neonatal period Benign familial neonatal epilepsy(bfne) Early myoclonic encephalopathy (EME) Ohtahara syndrome Infancy Epilepsy of infancy with migrating focal seizures West syndrome Myoclonic epilepsy in infancy (MEI) Benign infantile epilepsy Benign familial infantile epilepsy Dravet syndrome Myoclonic encephalopathy in nonprogressive disorders Childhood Febrile seizures plus (FS+) (can start in infancy) Panayiotopoulos syndrome Epilepsy with myoclonic atonic (previously astatic) seizures Benign epilepsy with centrotemporal spikes (BECTS) Autosomal-dominant nocturnal frontal lobe epilepsy (ADNFLE) Late onset childhood occipital epilepsy (Gastaut type) Epilepsy with myoclonic absences Lennox-Gastaut syndrome Epileptic encephalopathy with continuous spike-andwave during sleep (CSWS) b Landau-Kleffner syndrome (LKS) Childhood absence epilepsy (CAE) Adolescence Adult
Generalized seizures Tonic clonic(in any combination) Absence Typical Atypical Absence with special features Myoclonic absence Eyelid myoclonia Myoclonic Myoclonic Myoclonic atonic Myoclonic tonic Clonic Tonic Atonic Focal seizures Unknown Epileptic spasms Neonatal period Benign familial neonatal epilepsy(bfne) Early myoclonic encephalopathy (EME) Ohtahara syndrome Infancy Epilepsy of infancy with migrating focal seizures West syndrome Myoclonic epilepsy in infancy (MEI) Benign infantile epilepsy Benign familial infantile epilepsy Dravet syndrome Myoclonic encephalopathy in nonprogressive disorders Childhood Febrile seizures plus (FS+) (can start in infancy) Panayiotopoulos syndrome Epilepsy with myoclonic atonic (previously astatic) seizures Benign epilepsy with centrotemporal spikes (BECTS) Autosomal-dominant nocturnal frontal lobe epilepsy (ADNFLE) Late onset childhood occipital epilepsy (Gastaut type) Epilepsy with myoclonic absences Lennox-Gastaut syndrome Epileptic encephalopathy with continuous spike-andwave during sleep (CSWS) b Landau-Kleffner syndrome (LKS) Childhood absence epilepsy (CAE) Adolescence Adult For seizures Focal (previously partial ): the first clinical and electroencephalographic changes indicate initial activation of a system of neurons limited to a part of one cerebral hemisphere Generalized: the first clinical changes indicate initial involvement of both hemispheres For epilepsies Localization-related (focal, partial): epilepsies with focal seizures Generalized: epilepsies with generalized seizures Idiopathic: there is no underlying cause other than a possible Focal and generalized Etiology Focal seizures are conceptualized as originating at some point within networks limited to one hemisphere Generalized seizures are conceptualized as originating at some point within and rapidly engaging bilaterally distributed networks These terms were abandoned as overarching categories for classifying epilepsies per se, as many syndromes include both seizure types; they may still apply in some but not all instances Genetic: the epilepsy is, as best as understood, the direct result of a known or presumed genetic defect(s) in which seizures are the core symptom of the disorder. This attribution must be supported by specific forms of evidence Structural/metabolic: there is a distinct other structural or metabolic condition or disease that
Generalized seizures Tonic clonic(in any combination) Absence Typical Atypical Absence with special features Myoclonic absence Eyelid myoclonia Myoclonic Myoclonic Myoclonic atonic Myoclonic tonic Clonic Tonic Atonic Focal seizures Unknown Epileptic spasms Neonatal period Benign familial neonatal epilepsy(bfne) Early myoclonic encephalopathy (EME) Ohtahara syndrome Infancy Epilepsy of infancy with migrating focal seizures West syndrome Myoclonic epilepsy in infancy (MEI) Benign infantile epilepsy Benign familial infantile epilepsy Dravet syndrome Myoclonic encephalopathy in nonprogressive disorders Childhood Febrile seizures plus (FS+) (can start in infancy) Panayiotopoulos syndrome Epilepsy with myoclonic atonic (previously astatic) seizures Benign epilepsy with centrotemporal spikes (BECTS) Autosomal-dominant nocturnal frontal lobe epilepsy (ADNFLE) Late onset childhood occipital epilepsy (Gastaut type) Epilepsy with myoclonic absences Lennox-Gastaut syndrome Epileptic encephalopathy with continuous spike-andwave during sleep (CSWS) b Landau-Kleffner syndrome (LKS) Childhood absence epilepsy (CAE) Adolescence Adult For seizures Focal (previously partial ): the first clinical and electroencephalographic changes indicate initial activation of a system of neurons limited to a part of one cerebral hemisphere Generalized: the first clinical changes indicate initial involvement of both hemispheres For epilepsies Localization-related (focal, partial): epilepsies with focal seizures Generalized: epilepsies with generalized seizures Idiopathic: there is no underlying cause other than a possible Focal and generalized Etiology Focal seizures are conceptualized as originating at some point within networks limited to one hemisphere Generalized seizures are conceptualized as originating at some point within and rapidly engaging bilaterally distributed networks These terms were abandoned as overarching categories for classifying epilepsies per se, as many syndromes include both seizure types; they may still apply in some but not all instances Genetic: the epilepsy is, as best as understood, the direct result of a known or presumed genetic defect(s) in which seizures are the core symptom of the disorder. This attribution must be supported by specific forms of evidence Structural/metabolic: there is a distinct other structural or metabolic condition or disease that
Lägg upp 128-136-144-152-160 m på rundst 3,5 med 1 tråd av varje kvalitet. Sticka resår = 2 rm, 2 am i 5 cm. Byt till rundst 4,5. Nästa v stickas så här: Sticka de första 50-54-58-62-66 m slätst SAMTIDIGT som det minskas 6-6-8-8-8 m jämnt fördelat (= 44-48-50-54-58 m slätst), de nästa 14 m fortsätts i resår som förut, sedan stickas 50-54-58-62-66 m slätst SAMTIDIGT som det minskas 6-6-8-8-8 m jämnt fördelat (= 44-48-50-54-58 m slätst), de sista 14 m fortsätts i resår som förut. Det är nu 116-124-128-136-144 m på st. Sticka sedan med slätst och resår i sidorna. När arb mäter 10-11-12-13-14 cm, ökas det 1 m på varje sida av de 14 resårmaskorna i varje sida (= 4 m ökade), dessa m slätstickas. Upprepa ökningen när arb mäter 18-20-22-24-26 cm = 124-132-136-144-152 m. KONTROLLERA STICKFASTHETEN! När arb mäter 26-29-32-35-38 cm, maskas de mittersta 8 resårm i varje sida av för ärmhål (= 1 rm, 2 am, 2 rm, 2 am, 1 rm) = 108-116-120-128-136 m kvar på st. Lägg arb åt sidan och sticka ärmarna
Läkemedelsboken 2014 Paul Uvebrant & Ingrid Olsson Klassifikation av anfall Fokala anfall Fokalt anfall utan medvetandepåverkan Fokalt anfall med medvetandepåverkan Fokalt anfall med utveckling till ett bilateralt konvulsivt anfall Generaliserade anfall (med samtidig start i båda hemisfärerna) Tonisk-kloniska anfall Absenser Atypiska absenser Myokloniska anfall Toniska anfall Kloniska anfall Atoniska anfall Anfall som inte kan klassificeras
Dagens dikt
Man bör akta sej numera, särskilt inom psykiatri för att generalisera men du är, det anser vi en komplex analt astenisk, hypokondrisk och hysterisk, melankolisk lätt kolerisk pyknisk, leptosom atlet. Du är en typisk narcissistisk, maniskt, schizoid, autistisk och empatisk masochistisk substabil personlighet en kombinerad, avancerad Oedipuskomplexfixerad, och perverst oralt kastrerad vädur med en släng av DAMP.
Du blir bra som sjukvårds pamp!